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Tetrahydroacridine inhibits voltage-dependent Na(+) current in guinea-pig ventricular myocytes

  
@article{APS8360,
	author = {Wei- WANG and Yi-ping WANG and Guo-yuan HU},
	title = {Tetrahydroacridine inhibits voltage-dependent Na(+) current in guinea-pig ventricular myocytes},
	journal = {Acta Pharmacologica Sinica},
	volume = {25},
	number = {9},
	year = {2016},
	keywords = {},
	abstract = {AIM:
To study the effects of tetrahydroacridine (tacrine) on voltage-gated Na(+) channels in cardiac tissues.
METHODS:
Single ventricular myocytes were enzymatically dissociated from adult guinea-pig heart. Voltage-dependent Na(+) current was recorded using whole cell voltage-clamp technique.
RESULTS:
(1) Tacrine reversibly inhibited Na(+) current with an IC(50) value of 120 micromol/L (95 % confidence range: 108-133 micromol/L). (2) The inhibitory effects of tacrine on Na(+) current exhibited both a tonic nature and use-dependence. (3) Tacrine at 100 micromol/L caused a negative shift (about 10 mV) in the voltage-dependence of steady-state inactivation of Na(+) current, and retarded its recovery from inactivation, but did not affect its activation curve. (4) Intracellular application of tacrine significantly inhibited Na(+) current.
CONCLUSION:
In addition to blocking other voltage-gated ion channels, tacrine blocked Na(+) channels in guinea-pig ventricular myocytes. Tacrine acted as inactivation stabilizer of Na(+) channels in cardiac tissues.},
	issn = {1745-7254},	url = {http://www.chinaphar.com/article/view/8360}
}