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Bepridil inhibition of sodium current in rat hippocampal CA1 neurons.

  
@article{APS8256,
	author = {Hui-juan Hu and Dong-mei Yang and De-zhi Dai and Cai-hong Wu and Pei-ai Zhou},
	title = {Bepridil inhibition of sodium current in rat hippocampal CA1 neurons.},
	journal = {Acta Pharmacologica Sinica},
	volume = {20},
	number = {9},
	year = {2016},
	keywords = {},
	abstract = {AIM:
To study the effects of bepridil on sodium current in rat hippocampal neurons.
METHODS:
All experiments were performed on acutely isolated hippocampal pyramidal neurons by means of whole-cell patch-clamp techniques. Recording media contained ion channel blockers to allow the selective activation of voltage-dependent sodium currents.
RESULTS:
Bepridil reduced the amplitudes of sodium current in time- and concentration-dependent manners. The half-blocking time was about 10 min in bepridil 10 micromol.L-1, and IC50 was 2.6 (2.3-2.9) micromol.L-1. Bepridil 10 micromol.L-1 shifted the maximal activation of sodium current from -50 mV to -40 mV, and the characteristic voltage of inactivation from -71 mV to -89 mV without changing the slope factor.
CONCLUSION:
Bepridil blocked voltage-dependent sodium current of hippocampal CA1 neurons and might have therapeutic actions for ischemia-induced brain damage.},
	issn = {1745-7254},	url = {http://www.chinaphar.com/article/view/8256}
}