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Activation of p42/44 mitogen-activated protein kinase pathway in long-term potentiation induced by nicotine in hippocampal CA1 region in rats

  
@article{APS7546,
	author = {Jie Wang and Yuan-Bin Chen and Xiao-Nan Zhu and Ru-Zhu Chen},
	title = {Activation of p42/44 mitogen-activated protein kinase pathway in long-term potentiation induced by nicotine in hippocampal CA1 region in rats},
	journal = {Acta Pharmacologica Sinica},
	volume = {22},
	number = {8},
	year = {2016},
	keywords = {},
	abstract = {Aim: To investigate the relationship between activation of p42/44 mitogen-activated protein kinase (MAPK) pathway and hippocampal long term potentiation (LTP) induced by nicotine in area CA 1.
Methods: Extracellular recording of population spike (PS) was performed within the pyramidal cell layer of hippocampal area CA1 in vitro; Western blot analysis was employed to detect the active phosphorylated state and the total protein expression of p42/44 MAPK.
Results: PD98059 concentration-dependently (25 micromol/L, 50 micromol/L) attenuated the induction of LTP induced by nicotine 10 micromol/L; both p42 and p44 MAPK were activated with their total protein expression increasing in CA1 subregion in response to LTP induced by nicotine.
Conclusion: Activation of p42/44 MAPK pathway is required for hippocampal LTP induced by nicotine.},
	issn = {1745-7254},	url = {http://www.chinaphar.com/article/view/7546}
}