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Platelet activation by platelet aggregation factor from Eisenia foelide

   
@article{APS6445,
	author = {Rong Qing and Yuan-gong Zhou and Qin-qi Zhuang},
	title = {Platelet activation by platelet aggregation factor from Eisenia foelide},
	journal = {Acta Pharmacologica Sinica},
	volume = {15},
	number = {5},
	year = {2016},
	keywords = {},
	abstract = {A platelet activating factor from earthworm, Eisenia foelide (EPAF, 25.9 mumol.L-1), induced human platelet aggregation and 5-HT (maximal release of 89% at EPAF 74.1 mumol.L-1) was detected during this process. Neither creatine phosphate/creatine phosphate kinase (CP/CPK) nor aspirin completely inhibited the EPAF-induced platelet aggregation. In the presence of fibrinogen, EPAF (55.6 mumol.L-1) induced the aggregation of human platelet which had been thrombin-treated and degranulated. Results indicated that EPAF was a potent platelet agonist and the EPAF-induced platelet aggregation was ADP- and TXA2-independent.},
	issn = {1745-7254},	url = {http://www.chinaphar.com/article/view/6445}
}