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Electrophysiological effect of fluoxetine on Xenopus oocytes heterologously expressing human serotonin transporter

  
@article{APS3758,
	author = {Hong-wei Wang and Ci-zhen Li and Zhi-fang Yang and Yan-qian Zheng and Ying Zhang and Yuan-mou Liu},
	title = {Electrophysiological effect of fluoxetine on Xenopus oocytes heterologously expressing human serotonin transporter},
	journal = {Acta Pharmacologica Sinica},
	volume = {27},
	number = {3},
	year = {2016},
	keywords = {},
	abstract = {Aim:  To investigate the electrophysiological effect of fluoxetine on serotonin transporter.
Methods:  A heterologous expression system was used to introduce human serotonin transporter (hSERT) into  Xenopus oocytes. A 2-electrode voltage clamp technique was used to study the pharmacological properties of fluoxetine.
Results:  hSERT-expressing oocytes were perfused with 10  mol/L serotonin (5-HT) to induce hSERT-current. The 5-HT-induced hSERT currents were dose-dependently reversed by fluoxetine. The RC50 (concentration that achieved a 50% reversal) was approximately 3.12  mol/L. Fluoxetine took more time to combine with hSERT than 5-HT did, and it was also slow to dissociate from hSERT. This long-lasting effect of fluoxetine affected normal 5-HT transport. Fluoxetine significantly prolonged the time constant for 5-HT-induced hSERT current. These results might be used to explain the long-lasting anti-anxiety effect of fluoxetine in clinical practice, because it increases the concentration of 5-HT in the synaptic cleft by its enduring suppression of the function of 5-HT transporters.
Conclusion:  Fluoxetine inhibits 5-HT reuptake by competing with 5-HT and changing the normal dynamics of hSERT.},
	issn = {1745-7254},	url = {http://www.chinaphar.com/article/view/3758}
}