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Signaling profiles in HEK 293T cells co-expressing GLP-1 and GIP receptors

  
@article{APS10580,
	author = {Yu-zhe Wang and De-hua Yang and Ming-wei Wang},
	title = {Signaling profiles in HEK 293T cells co-expressing GLP-1 and GIP receptors},
	journal = {Acta Pharmacologica Sinica},
	volume = {43},
	number = {6},
	year = {2022},
	keywords = {},
	abstract = {Glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP) are regarded as ‘incretins’ working closely to regulate glucose homeostasis. Unimolecular dual and triple agonists of GLP-1R and GIPR have shown remarkable clinical benefits in treating type 2 diabetes. However, their pharmacological characterization is usually carried out in a single receptor-expressing system. In the present study we constructed a co-expression system of both GLP-1R and GIPR to study the signaling profiles elicited by mono, dual and triple agonists. We show that when the two receptors were co-expressed in HEK 293T cells with comparable receptor ratio to pancreatic cancer cells, GIP predominately induced cAMP accumulation while GLP-1 was biased towards β-arrestin 2 recruitment. The presence of GIPR negatively impacted GLP-1R-mediated cAMP and β-arrestin 2 responses. While sharing some common modulating features, dual agonists (peptide 19 and LY3298176) and a triple agonist displayed differentiated signaling profiles as well as negative impact on the heteromerization that may help interpret their superior clinical efficacies.},
	issn = {1745-7254},	url = {http://www.chinaphar.com/article/view/10580}
}